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[1]GUANG H , XIN W , ZUO C , et al. Prevalence of heart failure and left ventricular dysfunction in China: the China Hypertension Survey, 2012-2015. [J]. European journal of heart failure, 2019, 21(11): 1329-1337.
[2]ROSCA MG, TANDLER B, HOPPEL CL. Mitochondria in cardiac hypertrophy and heart failure [J]. J Mol Cell Cardiol, 2013, 5(5): 31-41
[3]FARGE G, FALKENBERG M. Organization of DNA in Mammalian Mitochondria [J]. Int J Mol Sci, 2019, 20(11): 2770.
[4]PATIL V, CUENIN C, CHUNG F, AGUILERA JRR, FERNANDEZ-JIMENEZ N, ROMERO-GARMENDIA I, BILBAO JR, CAHAIS V, ROTHWELL J, HERCEG Z. Human mitochondrial DNA is extensively methylated in a non-CpG context [J]. Nucleic Acids Res, 2019, 47(19): 10072-10085.
[5]THURAIRAJAH K, BRIGGS GD, BALOGH ZJ. The source of cell-free mitochondrial DNA in trauma and potential therapeutic strategies [J]. Eur J Trauma Emerg Surg, 2018, 44(3): 325-334.
[6]KACZMAREK A, VANDENABEELE P, KRYSKO DV. Necroptosis: the release of damage-associated molecular patterns and its physiological relevance [J]. Immunity, 2013, 38(2): 209-23.
[7]BLIKSØEN M, MARIERO LH, OHM IK, HAUGEN F, YNDESTAD A, SOLHEIM S, SELJEFLOT I, RANHEIM T, ANDERSEN G, AUKRUST P, VALEN G, VINGE LE. Increased circulating mitochondrial DNA after myocardial infarction [J]. Int J Cardiol, 2012, 158(1): 132-4.
[8]GYÖRGY B, SZABÓTG, PÁSZTÓI M, PÁL Z, MISJÁK P, ARADI B, LÁSZLÓV, PÁLLINGER E, PAP E, KITTEL A, NAGY G, FALUS A, BUZÁS EI. Membrane vesicles, current state-of-the-art: emerging role of extracellular vesicles [J]. Cell Mol Life Sci, 2011, 68(16): 2667-88.
[9]JANOWSKA-WIECZOREK A, WYSOCZYNSKI M, KIJOWSKI J, MARQUEZ-CURTIS L, MACHALINSKI B, RATAJCZAK J, RATAJCZAK MZ. Microvesicles derived from activated platelets induce metastasis and angiogenesis in lung cancer [J]. Int J Cancer, 2005, 113(5): 752-60.
[10]RECORD M, CARAYON K, POIROT M, SILVENTE-POIROT S. Exosomes as new vesicular lipid transporters involved in cell-cell communication and various pathophysiologies [J]. Biochim Biophys Acta, 2014, 1841(1): 108-20.
[11]BOUDREAU LH, DUCHEZ AC, CLOUTIER N, SOULET D, MARTIN N, BOLLINGER J, et al. Platelets release mitochondria serving as substrate for bactericidal group IIA-secreted phospholipase A2 to promote inflammation [J]. Blood, 2014, 124(14): 2173-2183.
[12]MAEDA A, FADEEL B. Mitochondria released by cells undergoing TNF-alpha-induced necroptosis act as danger signals [J]. Cell Death Dis, 2014(5): 1312.
[13]ZHU M, BARBAS AS, LIN L, SCHEUERMANN U, BISHAWI M, BRENNAN TV. Mitochondria Released by Apoptotic Cell Death Initiate Innate Immune Responses [J]. Immunohorizons, 2018, 2(11): 384-397.
[14]SHEPARD CR. TLR9 in MAFLD and NASH: At the Intersection of Inflammation and Metabolism [J]. Front Endocrinol(Lausanne), 2021(11): 613639.
[15]UEDA H, YAMAGUCHI O, TANEIKE M, AKAZAWA Y, WADA-KOBAYASHI H, SUGIHARA R, YORIFUJI H, NAKAYAMA H, OMIYA S, MURAKAWA T, SAKATA Y, OTSU K. Administration of a TLR9 Inhibitor Attenuates the Development and Progression of Heart Failure in Mice [J]. JACC Basic Transl Sci, 2019, 4(3): 348-363.
[16]GUESCINI M, GENEDANI S, STOCCHI V, AGNATI LF. Astrocytes and Glioblastoma cells release exosomes carrying mtDNA [J]. J Neural Transm(Vienna), 2010, 117(1): 1-4.
[17]YE W, TANG X, YANG Z, LIU C, ZHANG X, JIN J, LYU J. Plasma-derived exosomes contribute to inflammation via the TLR9-NF-κB pathway in chronic heart failure patients [J]. Mol Immunol, 2017, 8(7): 114-121.
[18]DUERR GD, HEINE A, HAMIKO M, ZIMMER S, LUETKENS JA, NATTERMANN J, RIEKE G, ISAAK A, JEHLE J, HELD SAE, WASMUTH JC, WITTMANN M, STRASSBURG CP, BROSSART P, COBURN M, TREEDE H, NICKENIG G, KURTS C, VELTEN M. Parameters predicting COVID-19-induced myocardial injury and mortality [J]. Life Sci, 2020, 26(4): 118400.
[19]LIU FY, FAN D, YANG Z, TANG N, GUO Z, MA SQ, MA ZG, WU HM, DENG W, TANG QZ. TLR9 is essential for HMGB1-mediated post-myocardial infarction tissue repair through affecting apoptosis, cardiac healing, and angiogenesis [J]. Cell Death Dis, 2019, 10(7): 480.
[20]OKA T, HIKOSO S, YAMAGUCHI O, TANEIKE M, TAKEDA T, TAMAI T, OYABU J, MURAKAWA T, NAKAYAMA H, NISHIDA K, AKIRA S, YAMAMOTO A, KOMURO I, OTSU K. Mitochondrial DNA that escapes from autophagy causes inflammation and heart failure [J]. Nature, 2012, 485(7397): 251-5.
[21]MANN DL. Inflammatory mediators and the failing heart: past, present, and the foreseeable future [J]. Circ Res, 2002, 91(11): 988-98.
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